Dying to entrain: Regulating ipRGC spacing

Filipe Pinto-Teixeira; Claude Desplan

doi:10.1016/j.devcel.2013.02.001

Dying to entrain: Regulating ipRGC spacing

Filipe Pinto-Teixeira, Claude Desplan

Research output: Contribution to journal › Short survey › peer-review

Abstract

In a recent issue of Neuron, Chen et al. (2013) show that apoptosis is required to ensure the even distribution of a class of retinal ganglion cells (ipRGCs), which sense luminance both intrinsically and through input from rods and cones. Disrupting apoptosis impairs photoentrainment mediated by rods/cones, but not that mediated by ipRGC-expressed melanopsin.

Original language	English (US)
Pages (from-to)	338-340
Number of pages	3
Journal	Developmental Cell
Volume	24
Issue number	4
DOIs	https://doi.org/10.1016/j.devcel.2013.02.001
State	Published - Feb 25 2013

ASJC Scopus subject areas

Molecular Biology
General Biochemistry, Genetics and Molecular Biology
Developmental Biology
Cell Biology

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10.1016/j.devcel.2013.02.001

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title = "Dying to entrain: Regulating ipRGC spacing",

abstract = "In a recent issue of Neuron, Chen et al. (2013) show that apoptosis is required to ensure the even distribution of a class of retinal ganglion cells (ipRGCs), which sense luminance both intrinsically and through input from rods and cones. Disrupting apoptosis impairs photoentrainment mediated by rods/cones, but not that mediated by ipRGC-expressed melanopsin.",

author = "Filipe Pinto-Teixeira and Claude Desplan",

year = "2013",

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AU - Pinto-Teixeira, Filipe

AU - Desplan, Claude

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N2 - In a recent issue of Neuron, Chen et al. (2013) show that apoptosis is required to ensure the even distribution of a class of retinal ganglion cells (ipRGCs), which sense luminance both intrinsically and through input from rods and cones. Disrupting apoptosis impairs photoentrainment mediated by rods/cones, but not that mediated by ipRGC-expressed melanopsin.

AB - In a recent issue of Neuron, Chen et al. (2013) show that apoptosis is required to ensure the even distribution of a class of retinal ganglion cells (ipRGCs), which sense luminance both intrinsically and through input from rods and cones. Disrupting apoptosis impairs photoentrainment mediated by rods/cones, but not that mediated by ipRGC-expressed melanopsin.

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JO - Developmental Cell

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Dying to entrain: Regulating ipRGC spacing

Abstract

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