TY - JOUR
T1 - Hst3p, a histone deacetylase, promotes maintenance of Saccharomyces cerevisiae chromosome III lacking efficient replication origins
AU - Irene, Carmela
AU - Theis, James F.
AU - Gresham, David
AU - Soteropoulos, Patricia
AU - Newlon, Carol S.
N1 - Funding Information:
We thank J. Boeke, M.M. Smith and D. Allis for providing plasmids, Dana Stein at the NJMS Flow Cytometry Core Laboratory for assistance with FACS analysis, Tongsheng Wang and Donna Storton for their assistance with the microarray processing, and Ann Dershowitz for fluctuation analysis. We thank also Drs. Michael Newlon, Vivian Bellofatto, Katsunori Sugimoto, Karl Drlica and all the members of the Newlon lab for helpful discussions of the manuscript. This work was supported by NIH Grant GM 35679 to C.S.N.
Publisher Copyright:
© 2015, The Author(s).
PY - 2016/2/1
Y1 - 2016/2/1
N2 - Long gaps between active replication origins probably occur frequently during chromosome replication, but little is known about how cells cope with them. To address this issue, we deleted replication origins from S. cerevisiae chromosome III to create chromosomes with long interorigin gaps and identified mutations that destabilize them [originless fragment maintenance (Ofm) mutations]. ofm6-1 is an allele of HST3, a sirtuin that deacetylates histone H3K56Ac. Hst3p and Hst4p are closely related, but hst4Δ does not cause an Ofm phenotype. Expressing HST4 under the control of the HST3 promoter suppressed the Ofm phenotype of hst3Δ, indicating Hst4p, when expressed at the appropriate levels and/or at the correct time, can fully substitute for Hst3p in maintenance of ORIΔ chromosomes. H3K56Ac is the Hst3p substrate critical for chromosome maintenance. H3K56Ac-containing nucleosomes are preferentially assembled into chromatin behind replication forks. Deletion of the H3K56 acetylase and downstream chromatin assembly factors suppressed the Ofm phenotype of hst3, indicating that persistence of H3K56Ac-containing chromatin is deleterious for the maintenance of ORIΔ chromosomes, and experiments with synchronous cultures showed that it is replication of H3K56Ac-containing chromatin that causes chromosome loss. This work shows that while normal chromosomes can tolerate hyperacetylation of H3K56Ac, deacetylation of histone H3K56Ac by Hst3p is required for stable maintenance of a chromosome with a long interorigin gap. The Ofm phenotype is the first report of a chromosome instability phenotype of an hst3 single mutant.
AB - Long gaps between active replication origins probably occur frequently during chromosome replication, but little is known about how cells cope with them. To address this issue, we deleted replication origins from S. cerevisiae chromosome III to create chromosomes with long interorigin gaps and identified mutations that destabilize them [originless fragment maintenance (Ofm) mutations]. ofm6-1 is an allele of HST3, a sirtuin that deacetylates histone H3K56Ac. Hst3p and Hst4p are closely related, but hst4Δ does not cause an Ofm phenotype. Expressing HST4 under the control of the HST3 promoter suppressed the Ofm phenotype of hst3Δ, indicating Hst4p, when expressed at the appropriate levels and/or at the correct time, can fully substitute for Hst3p in maintenance of ORIΔ chromosomes. H3K56Ac is the Hst3p substrate critical for chromosome maintenance. H3K56Ac-containing nucleosomes are preferentially assembled into chromatin behind replication forks. Deletion of the H3K56 acetylase and downstream chromatin assembly factors suppressed the Ofm phenotype of hst3, indicating that persistence of H3K56Ac-containing chromatin is deleterious for the maintenance of ORIΔ chromosomes, and experiments with synchronous cultures showed that it is replication of H3K56Ac-containing chromatin that causes chromosome loss. This work shows that while normal chromosomes can tolerate hyperacetylation of H3K56Ac, deacetylation of histone H3K56Ac by Hst3p is required for stable maintenance of a chromosome with a long interorigin gap. The Ofm phenotype is the first report of a chromosome instability phenotype of an hst3 single mutant.
KW - DNA replication
KW - Genome stability
KW - Histone H3 K56 acetylation
KW - SNP analysis
KW - Sirtuin
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U2 - 10.1007/s00438-015-1105-8
DO - 10.1007/s00438-015-1105-8
M3 - Article
C2 - 26319649
AN - SCOPUS:84955710494
SN - 1617-4615
VL - 291
SP - 271
EP - 283
JO - Molecular Genetics and Genomics
JF - Molecular Genetics and Genomics
IS - 1
ER -